I was looking at the internet. It just sat there, didn't do much.

this article got me thinking about Plant Sterols as Aphrodisiacs. Mostly because the article was about Plant Sterols acting as Aphrodisiacs in humans.

Have I got an herb for you!
Aphrodisiacs have been used worldwide since the dawn of human history. Researchers are just beginning to discover their chemical and physiological functions.
http://pubs.acs.org/subscribe/archive/ci...wartz.html

Quote:The plant "DAMIANA" contains "β-Sitosterol"
CAS# 84-46-5
Synonyms:
22,23-Dihydrostigmasterol,
Stigmast-5-en-3-ol,
β-Sitosterin


β-Sitosterol is widely distributed in the plant kingdom and found in Nigella sativa, pecans, Serenoa repens (saw palmetto), avocados, Cucurbita pepo (pumpkin seed), Pygeum africanum, cashew fruit, rice bran, wheat germ, corn oils, soybeans, sea-buckthorn, wolfberries, and Wrightia tinctoria.

But there was a molecule:
CAS# 491-80-5
Biochanin A
synonyms:

Quote:
BIOCHANIN A;
5,7-dihydroxy-3-(4-methoxyphenyl)-4h-1-benzopyran-4-on;
5,7-dihydroxy-3-(4-methoxyphenyl)-4H-1-Benzopyran-4-one;
APIGENIN 4-METHYL ETHER;
5,7-DIHYDROXY-4'-METHOXYISOFLAVONE;
5,7-DIHYDROXY-4-METHOXYISOFLAVONE;
Genistein 4′-methyl ether ;
5,7-dihydroxy-3-p-methoxyphenyl-4H-chromen-4-one

Biochanin A comes from RED CLOVER PLANT

Also had a "Spanish Fly" type description of:

Quote:Mechanism of Action:
It is the sex trigger for women’s use without color and scent and could easily dissolves in water, drink or alcohol. In a few minutes after taking, women would have itchy feeling in their vagina and would feel tingle on the inside of leg with hurried heartbeat and more and more private parts secretion.

Yohimbine Hypochloride
(not Yohimbe, but it comes from yohimbe, like 6% of yohimbe is Yohimbine Hypochloride, the other 94% is other stuff)


muira puama (Ptychopetalum olacoides), also known as “erection root”

Quote:Duke also recommends that women seeking to boost their sex drive and receptiveness to touch embrace dong quai, damiana, epimedium, fenugreek, ginger, and ginseng, “and that most ‘yin’ herb of folklore, wild yam”. Duke, who formulated the blend, calls it “Frigiditea”. His new book The Green Pharmacy Anti-Aging Prescriptions—Herbs, Foods, and Natural Formulas To Keep You Young

also, plant sterols that are recommended as human aphrodisiacs:

campesterol (CAS# 474-62-4, Campestanol; (24R)-Ergost-5-en-3β-ol) Banana, pomegranate, pepper, coffee, grapefruit, cucumber, onion, oat, potato, and lemon grass (citronella) are few examples of common sources containing campesterol at ~1-7 mg/100 mg of the edible portion. In contrast canola and corn oil contain as much as 16-100 mg/100mg.



stigmasterol (CAS# 83-48-7, Stigmasterin)


Stigmastanol CAS# 83-45-4,
aka
(3β)-Stigmastan-3-ol;
(3β,5α)-Stigmastan-3-ol;
β-Sitostanol;
Dihydrositosterin;
Dihydrositosterol;
Dihydro-β-sitosterol;
Fucostanol;
Spinastanol;
24α-Ethylcholestanol)
http://www.hmdb.ca/metabolites/HMDB00494


?other stigmastanol CAS#19466-47-8?



ergosterol (provitamin D2)
(ergosta-5,7,22-trien-3β-ol)
It is turned into viosterol by ultraviolet light, and is then converted into ergocalciferol, a form of vitamin D also known as D2

Don't these plant sterols and phytosterols look alot like some of the putative pheromone chemical structures?

It makes me wonder a little. Androtics Direct markets their Commercial Line and Putative PXXX series as using plant sterols to aid in diffusion. But IF they contain some of these plant sterols, could they in effect be saying that by mixing a putitive, with a well known plant sterol that acts as an aphrodisiac steroid, you can better bring out the aphrodisiac effect of the putative.

But what you may really see is, the aphrodisiac effect of the plant sterol, which is an aphrodisiac.

Stupid internet. I read an article, then another, then I put two peices of a puzzle together, then I post them as a coherent thought. This is probably why Androtics Direct erases/moderates my posts on their website sometimes and didn't send a free sample to me in my most recent order from them. I drink the stupid Kool-Aid, and then say, "Hey the blue fruit punch and the red fruit punch taste the same."

My "Blueberry Scent" that I created in May 2012 :

40 drops Damiana essential oil
10 drops Canadian Catnip essential oil (they say the Indian one doesn't work)
10 drops Neroli 5% essential oil

2,5 ml Iso-E Super
4 pellets Lauricidin
About 7 ml Perfumers Alcohol (fill it up)

Put it in a 10 ml spray and let it age a few days

The ladies like it, it lasts forever, smells a bit like blueberries.
Don't know about the effects though, I used it as a coverscent.

Edit : I only did 2 versions of it and it might be tried without the Neroli and with less Iso-E. It might very well have aphrodisiac effects, I just never tried it isolated...

For what it's worth; beta sitosterol is notable for behaving in the body like finesteride/propecia (finesteride is the stuff that is supposed to prevent hair loss.) In short, it prevents testosterone from converting into DHT. DHT is a a powerful androgen which also causes hair loss. (Merck seems to play down the benefits of DHT since they sell a pill which tries to prevent its creation, so be careful of popular sources on this topic.)

I've heard it said (not sure how reliable the information was) that some men's sex drive works off of DHT, some off of T and some off of both. If someone's body relied primarily on DHT, the stuff could kill their sex drive. If their sex drive relied on T, it would boost it. If it relied on both... who knows?

Are there certain instances where you'd WANT to reduce conversion of T to DHT for social reasons? Maybe. I'd be interested to hear that argument. Perhaps, you could cause someone to build up a stockpile of T which would be converted to DHT when the beta sitosterol wore off?

Though given that the primary conversion sites for T -> DHT are the scalp and prostate in men (And beta-sitosterol doesn't seem to even reach the prostate in significant quantities), I don't know if pheromonal effects would even interfere with its activity. I suppose that COULD change things entirely.

This could all be pointless speculation, of course. Let me know if your results say something else. I'm just trying to guess what the effects might be based on the published lit.

I tried yohimbe a while back as a pheromone and got no results. But that was a very early attempt and I looked mainly for self effects. Perhaps I didn't dilute it enough and that over-wrote the signal the way that too high of a dose can. Of course, I got no results taking the stuff orally either, so I could just be immune to it, personally. Or maybe I got a bad batch of the stuff. It'd be good to test on others who have benefited from taking the stuff orally. People who are personally immune to alpha androstenol seem less likely to get social benefits from it as well.



counteracting type I and type II isoforms of 5

Finasteride







Testosterone

Edti: You know, thinking it through and I really can't guess what the pheromonal effect of finesteride or beta sitosterol will be based on the systemic effect since I doubt much of the stuff will reach the scalp which seems to be beta sitosterol's primary target (or scalp and prostate for finesteride.)

11-Beta hydroxysteroid dehydrogenase inhibitors seem to work pheromonally. I'll have to see which tissues produce that. If they're far afield from the brain, perhaps beta-sitosterol will behave like it does in the body.

Is there any market for pheromones which prevent people from behaving sexually or that reduced sex drive? Maybe some women would favor that if they were getting unwanted attention. A sort of "down boy" mone... noone's ever asked for it, though. It would filter out a certain type of guy. The high testosterone types would be most strongly affected. There's an interesting social experiment...